Necrotising fasciitis, the flesh-eating disease, is rising in New Zealand, a new study shows. It is a severe bacterial infection commonly caused by Group A Streptococcus (GAS).
Necrotising fasciitis, the flesh-eating disease, is rising in New Zealand, a new study shows. It is a severe bacterial infection commonly caused by Group A Streptococcus (GAS). It results in the rapid destruction of soft tissue, with most severely affected people requiring extensive surgery and sometimes amputation of limbs to prevent infection from spreading.
University of Otago, Wellington researchers, Dr Dilip Das and Associate Professor Michael Baker, have shown that Necrotising fasciitis (NF) has risen from fewer than 10 cases a year in 1990 to more than 70 cases a year in 2006. The study has just been published in the international Journal of Infection.
The study identified 812 NF cases between 1990 and 2006, of whom 148 died, giving a 21 per cent fatality risk.
“Although this disease remains uncommon, it’s feared because of its speed of onset, high fatality rate, and the severe tissue damage its causes,” says Associate Professor Baker.
“This infection can follow from just a minor skin injury or wound. If anyone is suffering severe local pain and fever following even a small skin wound they should immediately see a doctor,” he says.
“The pain is caused by a very rapid spread of the infection into deep soft tissue and is not always obvious from first inspection of the skin. Rapid treatment is absolutely vital to save lives with NF.”
Advertisement
The rise in NF is part of a worrying increase in serious infectious diseases in New Zealand, says Associate Professor Baker. This paper follows a release from the University of Otago, Wellington earlier in the year (April 27) that identified nearly a 100 per cent increase in hospitalisations for serious skin infections in children since 1990.
Advertisement
He says the causes for this increase need further research to identify prevention measures.
This study was funded by the Ministry of Health.
Source-Medindia