Autism is directly affected by the food we eat. Mineral deficiencies, inability to remove toxins from the body and use of high fructose corn syrup may fuel autism.
Autism is a disorder of the neural development that is characterized by the disturbed social communication and interaction. There is a repetitive and restricted behavior. Diet plays a role in autism and care should be taken to make food choices. According to the Center for Disease Control and Prevention, 1 out of every 88 children has autism spectrum disorder (ASD). This is a serious concern.
Renee Dufault et al conducted a study to find out the influence of diet on autism. The study revealed that typical American diet, owing to mineral deficiencies and high fructose content, may trigger autism.
Dufault devised a new scientific approach, “macroepigenetic” to determine the influence of dietary factors like fructose on the body and chronic disease development.
Macroepigenetics studies the role of various environmental, nutritional and genetic factors in contributing to any specific ailment.
The researchers used the macroepigenetic approach to analyze the differences between exposure to toxic substances and diet and their impact on development of autism between American and Italian population.
Dufault and his fellow workers said that the occurrence of autism in USA and Italy was not affected by mercury exposure to fish, vaccines or dental amalgams. The study highlighted the presence of high fructose corn syrup (HFCS) in the commonly consumed American food (cookies, cereals, breads, crackers, etc.). HFCS creates a deficiency of zinc which in turn interferes with the elimination of cadmium, arsenic and mercury, thereby causing a negative impact on the neurodevelopment.
The blood homocysteine level increases with exposure to inorganic mercury and intake of high fructose corn syrup. This adversely affects the neurodevelopment and the occurrence of autism.
Reference: A macroepigenetic approach to identify factors responsible for the autism epidemic in the United States; Renee Dufault et al; Clinical Epigenetics 2012
Source-Medindia